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As a few have pointed out, the claim about lowering cortisol is contextual here to hypercortisolism. There is more nuance in other cases where cortisol may mildly increase, or increase at first and then return to baseline.

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Apparently this joke was too obscure or too veiled. I was referring to the pictured meme. Explanation in quoted tweet. x.com/KetoCarnivore/…

D12451: what's a good estimate of percent linoleic acid by calories? 10%? Peter I-live-in-the-paradoxes InNorfolk Tucker Goodrich

It’s happening! L. Amber O'Hearn on vitamin A: mostly-fat.com/2025/06/hyperv…

"This case report demonstrates that even in difficult social circumstances and with limited professional support it is potentially possible to implement a ketogenic metabolic therapy and put serious mental illness into remission." frontiersin.org/journals/nutri…

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The broken logic Raphi points out here should be patently obvious. I've also pointed this problem out to them half a dozen times over the last couple years, but the originators either don't get it or don't admit it and I'm not sure which is worse.

Oof. It hadn't occurred to me that the brain allowing in PUFA for DHA and ARA also means it can allow in linoleic acid. From the first paper in quote, that LA ends up mostly β-oxidized—not great in the brain!

What's the latest and greatest in home uric acid testing?

Somewhat surprising to me, because one theory for Neanderthal extinction was inability to harvest enough fat to cope with mega-fauna extinction (Miki Ben-Dor), but perhaps this hadn’t happened yet, or there was too much competition. Link to paper in press release.

“The hypothesis that linoleic acid generates insulin resistance promptly, as a direct effect of the generation of reactive aldehydes formed from linoleic acid in the bloodstream, is not supported by either of the Carpentier papers discussed here. “Far more plausible is the

"[T]he (inappropriate) fatty acid supply to insulin sensitive cells in obesity *requires* insulin resistance."