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In case you think that Nature Neuroscience doesn't publish negative results...

Directed by David Lynch.

Did you know it takes an average of 8 years to diagnose #BipolarDisorder? Newly published research from the Psychiatric Genomics Consortium (bit.ly/3JkuokY) aims to improve this by learning more about the condition’s biology. Now in Nature: go.nature.com/3PJhi3W

Our new findings from the PGC Bipolar working group published today: great team work!

Mount Sinai’s Dr. Niamh Mullins and an international team have identified 298 genetic regions linked to bipolar disorder, a 4x increase in our understanding of its genetic risk factors. This groundbreaking study spanned diverse ancestries and revealed 36 key genes—paving the way

.Niamh Mullins, et al have identified 298 genetic regions linked to #bipolardisorder. Groundbreaking study spanned diverse ancestries & revealed 36 key genes-paving way for innovative tmts, new drug targets, & precision medicine approaches. Full study👉mshs.co/3CherMq

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Vincent van Gogh.

Van Gogh with animated paintings…

Greek Farmer Accidentally Discovers 3400 year old Minoan Tomb Hidden Under Olive Grove - Lerapetra, Crete, Greece 🇬🇷 The untouched Bronze Age tomb and the skeletons inside will hopefully provide archaeologists with information about the mysterious Minoan civilization. It is a

🔬 Recent work lead by Maria Koromina identified 17 likely causal SNPs for BD & linked them to 23 genes involved in neurotransmission & neurodevelopment (e.g., SCN2A, TRANK1, DCLK3). Findings could improve polygenic risk scores across diverse populations🧠 #BipolarDisorder #Genetics

Join ⁦⁦AI and Human Health at Mount Sinai⁩ ⁦HPI.MS⁩ & ⁦Charles Bronfman Inst for Personalized Medicine⁩ for The Inaugural Mount SinAI Retreat on May 19! A full day of… 🧠 Inspiring talks + panels 🎤 Keynote by Dean Charney 🤖 AI tech demos (robots + AR goggles!) 🧬 Posters + cocktail hour

Beautiful work demonstrating how common variants modify the penetrance of rare variants in both children and adults with telomere biology disorders (TBDs). According to the liability threshold model, rare and common variants additively raise disease risk beyond a threshold,

Past eQTL studies have mostly focused on one gene at a time. But in the human genome, genes often occur in clusters and tend to be co-regulated by shared mechanisms. So why not consider neighboring genes together when mapping eQTLs? A new preprint tests this idea and introduces

Admit it when you're wrong and shut up when you're right. This is a simple way to drastically improve the quality of your relationships

What specific genetic variants actually cause bipolar disorder, and can we identify them among the thousands of associated DNA changes found in genome-wide studies?Nature Neuroscience Mount Sinai Psychiatry Mount Sinai Genetics "Fine-mapping genomic loci refines bipolar disorder risk genes"

Fine-mapping genomic loci refines bipolar disorder risk genes Maria Koromina, Niamh Mullins nature.com/articles/s4159…

A comprehensive fine-mapping of bipolar disorder GWAS signals by Maria Koromina et al. in Nature Neuroscience Great work with beautiful visualizations! nature.com/articles/s4159…

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