NetaRosezweig (@netarose11) 's Twitter Profile
NetaRosezweig

@netarose11

Neuroimmunology

ID: 1004773177862615041

calendar_today07-06-2018 17:12:48

26 Tweet

18 Followers

41 Following

♤בת תמר♧ (@kqdwsh) 's Twitter Profile Photo

תהיות אתה יודע מה היופי. שגם אם דעתי שונה משלך, אלמד להקשיב ולקבלה. שתזכה להתחדש כל פעם מחדש לטובה🌷 קודם כל לחתור להחזיר את כל השבויים. ולהפוך את רחל אדרי לראש הממשלה🙃

Oleg Butovsky (@olegbutovsky) 's Twitter Profile Photo

Dear Friends and Colleagues, A devastating terror shook Israel, targeting kibbutzim. This tragedy deeply saddens us.   In response, our Boston community, alongside a local Israeli non-profit organization, has launched a campaign to aid the affected families in their recovery.

Oleg Butovsky (@olegbutovsky) 's Twitter Profile Photo

During this darkest hour, the Sun encourages you to join us in extending a helping hand to the Israeli kibbutzim as they begin their long journey to recovery. kibbutzim-appeal.postedstuff.com/V2-f7Sd-WIBt-J…

Oleg Butovsky (@olegbutovsky) 's Twitter Profile Photo

Dear Harvard Community and Alumni, We, the Israeli students at Harvard College, are reaching out to you during a time of profound grief and urgency. Over the weekend, the community of Kfar Azza, situated right next to the Gaza border, experienced an unimaginable tragedy.

Alzforum (@alzforum) 's Twitter Profile Photo

APOE4 locks microglia in a state of homeostasis, preventing them from clearing amyloid plaque or beckoning astrocytes to help. Deleting APOE4 stirred microglia to dispose of plaques and tau tangles. Brigham and Women's Hospital Mayo Clinic HKUST ow.ly/YwlG50Q18O9

John Lukens (@lukensjohnr) 's Twitter Profile Photo

Sex-dependent APOE4 neutrophil–microglia interactions drive cognitive impairment in Alzheimer’s disease Nature Medicine nature.com/articles/s4159…

Oleg Butovsky (@olegbutovsky) 's Twitter Profile Photo

Our new work published today @NatrureMedicine nature.com/articles/s4159… The work led by Neta Rosenzweig (NetaRosezweig) and Kilian Kleemann (@KilianKleemann). Thanks to our collaborators Bart Eggen, Blurton-Jones Lab, @ReisaSperling Howard L. Weiner, MD Roni Nowarski

Ghazaleh Eskandari-Sedighi (@ghazalehesk) 's Twitter Profile Photo

Another tour de force from Oleg Butovsky lab further expanding our understanding of the APOE biology in Alzheimer’s disease. Congrats to the entire team of authors!

Oleg Butovsky (@olegbutovsky) 's Twitter Profile Photo

Our new work published today @NatrureMedicine nature.com/articles/s4159… The work led by Neta Rosenzweig (NetaRosezweig) and Kilian Kleemann (@KilianKleemann). Thanks to our collaborators Bart Eggen, Blurton-Jones Lab, @ReisaSperling Howard L. Weiner, MD Roni Nowarski

Nat Rev Neurology (@natrevneurol) 's Twitter Profile Photo

Sex-dependent APOE4 neutrophil–microglia interactions drive cognitive impairment in Alzheimer disease nature.com/articles/s4159…

Nature Medicine (@naturemedicine) 's Twitter Profile Photo

APOE4-dependent neutrophil-microglia interactions drive cognitive impairment in female individuals with Alzheimer’s disease via IL17F ButovskyLab, Oleg Butovsky, NetaRosezweig, @KilianKleemann #neutrophils, #microglia, #IL-17 nature.com/articles/s4159…

Neural Cell News (@neuralcell) 's Twitter Profile Photo

This work led by Neta Rosenzweig (NetaRosezweig), @KilianKleemann, & Thomas Rust provides a translational basis to target IL-17F in APOE ε4 female carriers with cognitive impairment. 🧠 Congratulations to all the authors! Oleg Butovsky Bart Eggen go.nature.com/4eZef2H

This work led by Neta Rosenzweig (<a href="/NetaRose11/">NetaRosezweig</a>), @KilianKleemann, &amp; Thomas Rust provides a translational basis to target IL-17F in APOE ε4 female carriers with cognitive impairment. 🧠 

Congratulations to all the authors! <a href="/OlegButovsky/">Oleg Butovsky</a> <a href="/EggenBart/">Bart Eggen</a>

go.nature.com/4eZef2H
NetaRosezweig (@netarose11) 's Twitter Profile Photo

Sex-Dependent APOE4 Neutrophil–Microglia Interactions Drive Cognitive Impairment in Alzheimer’s Disease stemcellsciencenews.com/newsletter/228… via Science, Space & Robots News

Brigham and Women’s Research (@brighamresearch) 's Twitter Profile Photo

Research Spotlight: Uncovering How a Cellular Miscommunication Leads to Cognitive Impairment in Female Patients with Alzheimer’s Disease buff.ly/4d1NmJT ButovskyLab Oleg Butovsky NetaRosezweig @KilianKleemann

Nature Medicine (@naturemedicine) 's Twitter Profile Photo

APOE4-dependent neutrophil-microglia interactions drive cognitive impairment in female individuals with Alzheimer’s disease via IL17F ButovskyLab, Oleg Butovsky, NetaRosezweig, @KilianKleemann #neutrophils, #microglia, #IL-17 nature.com/articles/s4159…

Eric Topol (@erictopol) 's Twitter Profile Photo

Common immune system mediated threads of Alzheimer's disease and age-related macular degeneration, a new open-access review cell.com/immunity/abstr… Immunity

Common immune system mediated threads of Alzheimer's disease and age-related macular degeneration, a new open-access review
cell.com/immunity/abstr… <a href="/ImmunityCP/">Immunity</a>
Immunity (@immunitycp) 's Twitter Profile Photo

Online now: Alzheimer’s disease and age-related macular degeneration: Shared and distinct immune mechanisms dlvr.it/TKZSH4

Oleg Butovsky (@olegbutovsky) 's Twitter Profile Photo

Thrilled to share invited review with the brilliant NetaRosezweig! in the Immunity special issue on neuroimmune interactions. We explore shared immune pathways in Alzheimer’s & AMD, and chart new directions for cross-disease therapeutic strategies. cell.com/immunity/fullt…

Thrilled to share invited review with the brilliant <a href="/NetaRose11/">NetaRosezweig</a>! in the <a href="/ImmunityCP/">Immunity</a> special issue on neuroimmune interactions. We explore shared immune pathways in Alzheimer’s &amp; AMD, and chart new directions for cross-disease therapeutic strategies. cell.com/immunity/fullt…
Danny Saban (@sabanlab) 's Twitter Profile Photo

organoids, while a powerful tool for studying human biology, lack immune systems, which is a significant limitation. NIH prioritized organoids to model human tissues and diseases in vitro. However, their lack of an immune system hinders their ability to fully recapitulate human