Hanrui Zhang (@hanrui_zhang) 's Twitter Profile
Hanrui Zhang

@hanrui_zhang

Assistant Professor @Columbia_CGP @Columbia University | Technical Editor @atvbahajournals | Macrophage Biology | Functional Genomics | CRISPR

ID: 907419526899388416

linkhttps://hanruizhang.github.io/zhanglab/ calendar_today12-09-2017 01:44:09

1,1K Tweet

1,1K Followers

818 Following

Peter Sims (@psimslab) 's Twitter Profile Photo

Excited to share our CITE-seq atlas of the aging human immune system across tissues. Great collaboration with Donna Farber, Yosef Lab, Teichlab, Jo Jones Cambridge University and partnership with CZI Science! nature.com/articles/s4159…

ATVB: An AHA Journal (@atvbahajournals) 's Twitter Profile Photo

🚀 Just dropped! The latest blog from #ATVB_Net is live—featuring fresh insights, expert perspectives, and the latest in arteriosclerosis, vascular biology, and thrombosis research. 🔗 Dive in: ahajrnls.org/3SUzucy Stay curious. Stay informed. #ATVB #AHAJournals

Babak Razani, MD, PhD Laboratory (@razanilab) 's Twitter Profile Photo

LIPA locus #SNP (#Lysosome Acid Lipase gene) long known as #CVD risk. Cool work by Fang Li of Hanrui Zhang lab in European Society of Cardiology Journals uses high-tech functional #genomics to uncover mechanism backed by mouse model. Thanks for including us in the journey. academic.oup.com/eurheartj/adva…

ATVB: An AHA Journal (@atvbahajournals) 's Twitter Profile Photo

Check out the latest from #ATVB_net! On April 23, 2025, the ATVB Women's Leadership Committee (WLC) hosted its highly anticipated annual Mentor of Women Award Luncheon at the Vascular Discovery Conference in Baltimore. 🔗 ahajrnls.org/4g9GPiM

Moritz von Scheidt (@moritzscheidt) 's Twitter Profile Photo

In >8,600 CAD patients, CHIP was linked to higher mortality, visualized in plaque macrophages, and associated with plaque vulnerability. We uncovered a novel mechanism where TET2-CHIP drives lipid uptake & inflammation—making it a targetable source of residual risk. @DZHK_germany

In >8,600 CAD patients, CHIP was linked to higher mortality, visualized in plaque macrophages, and associated with plaque vulnerability. We uncovered a novel mechanism where TET2-CHIP drives lipid uptake & inflammation—making it a targetable source of residual risk. @DZHK_germany
Mani Subramanian 🫀 (@mani_cvd) 's Twitter Profile Photo

🚨Our Circulation Research study reveals that impaired macrophage DNase secretion sustains NET (DNA webs) accumulation, which disrupts dead cell clearance and destabilises atherosclerotic plaques.❤️‍🩹 British Heart Foundation William Harvey Research Institute Barts and The London, Queen Mary Queen Mary University of London ahajournals.org/doi/10.1161/CI…